Authors identify capsaicin receptor TRPV1 as a key regulator of Ca2+ entry into axoplasm that is required for developmental degeneration modeled by NGF withdrawal from sensory neurons of the dorsal root ganglion in vitro.
By Jeffery L. Twiss, M.D., Ph.D.
March 7, 2019

Authors identify capsaicin receptor TRPV1 as a key regulator of Ca2+ entry into axoplasm that is required for developmental degeneration modeled by NGF withdrawal from sensory neurons of the dorsal root ganglion in vitro.

Authors found that experimentally induced stroke causes a secondary wave of brain tissue inflammation that shares some molecular and morphological characteristics with atherosclerosis.
By Matthew Davidson, PhD
February 14, 2019

Authors found that experimentally induced stroke causes a secondary wave of brain tissue inflammation that shares some molecular and morphological characteristics with atherosclerosis.

Authors describes an unexpected effect of in utero electroporation of plasmid DNA on the distribution of microglia within the developing forebrain.
By Orly Reiner
January 24, 2019

Authors describe an unexpected effect of in utero electroporation of plasmid DNA on the distribution of microglia within the developing forebrain.

Dr. Jacqueline Barker tells the story about her first-author eNeuro paper that showed that action-outcome relationships are differentially encoded by the infralimbic prefrontal cortex during goal-directed and habitual behaviors.
By Rosalind S.E. Carney, DPhil
January 10, 2019

Dr. Jacqueline Barker tells the story about her first-author eNeuro paper that showed that action-outcome relationships are differentially encoded by the infralimbic prefrontal cortex during goal-directed and habitual behaviors.

eNeuro’s Top Downloaded Papers Of 2018!
By SfN Staff
December 20, 2018

Check out the top 10 most downloaded articles published in eNeuro in 2018.

Figure 1. Leeches that recover crawling behavior have established regrowth of afferent input to the lead ganglion. (A) In leeches that recover crawling behavior after injury, Neurobiotin labels fibers in the M3 nerve, which project into the M3 ganglion. (B) In leeches that do not recover crawling behavior after injury, Neurobiotin-labeled fibers are not observed in the M3 ganglion. (Figure 8 in Puhl et al. 2018.)
By Rosalind S.E. Carney, DPhil
December 13, 2018

Authors demonstrate that in the absence of descending cephalic input, locomotor recovery in the leech is achieved by a switch to dependence on afferent information from peripheral nerves in the body wall.

Authors addressed the question 'what, if anything, is rodent prefrontal cortex?' by carrying out a survey of prefrontal cortex researchers and uncovered points of agreement as well as differences in regard to what brain areas constitute "prefrontal cortex" in rodents."
By Mark G. Baxter
December 5, 2018

Authors addressed the question 'what, if anything, is rodent prefrontal cortex?' by carrying out a survey of prefrontal cortex researchers and uncovered points of agreement as well as differences in regard to what brain areas constitute "prefrontal cortex" in rodents."

Authors show that axons of inhibitory neurons have distinctive structural and molecular features that contrast with those of the majority of myelinated excitatory axons in human neocortex.
By Rosalind S.E. Carney, DPhil
November 29, 2018

Authors show that axons of inhibitory neurons have distinctive structural and molecular features that contrast with those of the majority of myelinated excitatory axons in human neocortex.

By Rosalind S.E. Carney, DPhil
October 31, 2018

Authors demonstrate that capsaicin, a TRPV1 agonist, activates a pro-axon growth program, suggesting an approach for enhancing axon regeneration in selective populations of neurons.

Authors functionally defined immediate-early astrocytes (ieAstrocytes) as an in vivo astrocyte population, modulated by S1P signaling, in a mouse model of multiple sclerosis.
By Rosalind S.E. Carney, DPhil
October 15, 2018

Authors functionally defined immediate-early astrocytes (ieAstrocytes) as an in vivo astrocyte population, modulated by S1P signaling, in a mouse model of multiple sclerosis.